Cover: Flowers from an Arabidopsis double mutant, ibm1 ldl2, which shows suppression of developmental defects of the ibm1 single mutant. The ibm1 mutant ectopically accumulates histone H3K9 methylation and DNA methylation in the internal regions (bodies) of active genes and causes developmental defects; the ldl2 mutation suppresses these developmental defects without affecting the repressive chromatin marks. Genetic interaction between IBM1 and LDL2 illuminates the roles of gene‐body chromatin modifications in epigenome differentiation. From Soichi Inagaki, Tetsuji Kakutani and colleagues: Gene‐body chromatin modification dynamics mediate epigenome differentiation in Arabidopsis. For details, see the Article on p 970. Photo by Soichi Inagaki.